Autoimmun hepatitis forudgået af hepatitis A-infektion

Anne Grave Pedersen; Jacob Juel; Mogens Vyberg; Søren Schou Olesen; Jesper Bach Hansen

Autoimmun hepatitis forudgået af hepatitis A-infektion

Anne Grave Pedersen, Jacob Juel, Mogens Vyberg, Søren Schou Olesen, Jesper Bach Hansen

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › peer review

Abstract

Autoimmune hepatitis is characterized by unresolving inflammation and fibrosis of the liver, which untreated may lead to cirrhosis. The pathogenesis remains unclear and reflects a complex interaction between autoantigens, genetic predisposition, triggering factors and immune regulation. In rare cases, acute hepatitis A has been suspected as a triggering factor. We describe a case of a 66-year-old woman who two months after a resolving acute hepatitis A presented with fatigue, pale stools and elevated levels of transaminases. A liver biopsy showed histological changes compatible with autoimmune hepatitis.

Bidragets oversatte titel	Autoimmune hepatitis preceded by hepatitis A
Originalsprog	Dansk
Artikelnummer	V12140669
Tidsskrift	Ugeskrift for Laeger
Vol/bind	177
Udgave nummer	20A
Antal sider	2
ISSN	0041-5782
Status	Udgivet - 23 mar. 2015

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Citationsformater

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AU - Pedersen, Anne Grave

AU - Juel, Jacob

AU - Vyberg, Mogens

AU - Olesen, Søren Schou

AU - Hansen, Jesper Bach

PY - 2015/3/23

Y1 - 2015/3/23

N2 - Autoimmune hepatitis is characterized by unresolving inflammation and fibrosis of the liver, which untreated may lead to cirrhosis. The pathogenesis remains unclear and reflects a complex interaction between autoantigens, genetic predisposition, triggering factors and immune regulation. In rare cases, acute hepatitis A has been suspected as a triggering factor. We describe a case of a 66-year-old woman who two months after a resolving acute hepatitis A presented with fatigue, pale stools and elevated levels of transaminases. A liver biopsy showed histological changes compatible with autoimmune hepatitis.

AB - Autoimmune hepatitis is characterized by unresolving inflammation and fibrosis of the liver, which untreated may lead to cirrhosis. The pathogenesis remains unclear and reflects a complex interaction between autoantigens, genetic predisposition, triggering factors and immune regulation. In rare cases, acute hepatitis A has been suspected as a triggering factor. We describe a case of a 66-year-old woman who two months after a resolving acute hepatitis A presented with fatigue, pale stools and elevated levels of transaminases. A liver biopsy showed histological changes compatible with autoimmune hepatitis.

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SN - 0041-5782

VL - 177

JO - Ugeskrift for Laeger

JF - Ugeskrift for Laeger

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Autoimmun hepatitis forudgået af hepatitis A-infektion

Abstract

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