Metabonomics identifies early molecular changes associated with progression into postoperative hypoxemia in cardiac surgery patient: a human model that can provide new insights into the pathophysiology of acute lung injury and potentially identify specific biomarkers of lung tissue injury

Bodil Steen Rasmussen; Raluca Maltesen; Munsoor Hanifa; Shona Pedersen; Søren Risom Kristensen; Reinhard Wimmer

doi:10.1186/s40635-016-0098-x

Metabonomics identifies early molecular changes associated with progression into postoperative hypoxemia in cardiac surgery patient: a human model that can provide new insights into the pathophysiology of acute lung injury and potentially identify specific biomarkers of lung tissue injury

Bodil Steen Rasmussen, Raluca Maltesen, Munsoor Hanifa, Shona Pedersen, Søren Risom Kristensen, Reinhard Wimmer

Research output: Contribution to journal › Conference abstract in journal › Research › peer-review

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Original language	English
Article number	A22
Journal	Intensive Care Medicine Experimental
Volume	4
Issue number	Suppl. 1
Pages (from-to)	13
ISSN	2197-425X
DOIs	https://doi.org/10.1186/s40635-016-0098-x
Publication status	Published - 2016
Event	Annual Congress of the European Society of Intensive Care Medicine, ESICM LIVES - Milan, Italy Duration: 1 Oct 2016 → 5 Oct 2016 Conference number: 29

Conference

Conference	Annual Congress of the European Society of Intensive Care Medicine, ESICM LIVES
Number	29
Country/Territory	Italy
City	Milan
Period	01/10/2016 → 05/10/2016

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10.1186/s40635-016-0098-x

Open Access abstractsFinal published version, 12.4 MB

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1 PhD thesis

NMR-Based Metabolomics in Kidney and Lung Disease: investigating metabolite biomarkers in a rat chronic kidney disease model and in human lung injury
Hanifa, M. A., 2020, Aalborg Universitetsforlag. 117 p.
Research output: PhD thesis

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Rasmussen, B. S., Maltesen, R., Hanifa, M., Pedersen, S., Kristensen, S. R., & Wimmer, R. (2016). Metabonomics identifies early molecular changes associated with progression into postoperative hypoxemia in cardiac surgery patient: a human model that can provide new insights into the pathophysiology of acute lung injury and potentially identify specific biomarkers of lung tissue injury. Intensive Care Medicine Experimental, 4(Suppl. 1), 13. Article A22. https://doi.org/10.1186/s40635-016-0098-x

Rasmussen, Bodil Steen ; Maltesen, Raluca ; Hanifa, Munsoor et al. / Metabonomics identifies early molecular changes associated with progression into postoperative hypoxemia in cardiac surgery patient : a human model that can provide new insights into the pathophysiology of acute lung injury and potentially identify specific biomarkers of lung tissue injury. In: Intensive Care Medicine Experimental. 2016 ; Vol. 4, No. Suppl. 1. pp. 13.

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title = "Metabonomics identifies early molecular changes associated with progression into postoperative hypoxemia in cardiac surgery patient: a human model that can provide new insights into the pathophysiology of acute lung injury and potentially identify specific biomarkers of lung tissue injury",

author = "Rasmussen, {Bodil Steen} and Raluca Maltesen and Munsoor Hanifa and Shona Pedersen and Kristensen, {S{\o}ren Risom} and Reinhard Wimmer",

year = "2016",

doi = "10.1186/s40635-016-0098-x",

language = "English",

volume = "4",

pages = "13",

journal = "Intensive Care Medicine Experimental",

issn = "2197-425X",

publisher = "Springer",

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Rasmussen, BS, Maltesen, R, Hanifa, M , Pedersen, S , Kristensen, SR & Wimmer, R 2016, 'Metabonomics identifies early molecular changes associated with progression into postoperative hypoxemia in cardiac surgery patient: a human model that can provide new insights into the pathophysiology of acute lung injury and potentially identify specific biomarkers of lung tissue injury', Intensive Care Medicine Experimental, vol. 4, no. Suppl. 1, A22, pp. 13. https://doi.org/10.1186/s40635-016-0098-x

Metabonomics identifies early molecular changes associated with progression into postoperative hypoxemia in cardiac surgery patient: a human model that can provide new insights into the pathophysiology of acute lung injury and potentially identify specific biomarkers of lung tissue injury. / Rasmussen, Bodil Steen; Maltesen, Raluca; Hanifa, Munsoor et al.
In: Intensive Care Medicine Experimental, Vol. 4, No. Suppl. 1, A22, 2016, p. 13.

Research output: Contribution to journal › Conference abstract in journal › Research › peer-review

TY - ABST

T1 - Metabonomics identifies early molecular changes associated with progression into postoperative hypoxemia in cardiac surgery patient

T2 - Annual Congress of the European Society of Intensive Care Medicine, ESICM LIVES

AU - Rasmussen, Bodil Steen

AU - Maltesen, Raluca

AU - Hanifa, Munsoor

AU - Pedersen, Shona

AU - Kristensen, Søren Risom

AU - Wimmer, Reinhard

N1 - Conference code: 29

PY - 2016

Y1 - 2016

U2 - 10.1186/s40635-016-0098-x

DO - 10.1186/s40635-016-0098-x

M3 - Conference abstract in journal

SN - 2197-425X

VL - 4

SP - 13

JO - Intensive Care Medicine Experimental

JF - Intensive Care Medicine Experimental

IS - Suppl. 1

M1 - A22

Y2 - 1 October 2016 through 5 October 2016

ER -

Rasmussen BS, Maltesen R, Hanifa M , Pedersen S , Kristensen SR , Wimmer R. Metabonomics identifies early molecular changes associated with progression into postoperative hypoxemia in cardiac surgery patient: a human model that can provide new insights into the pathophysiology of acute lung injury and potentially identify specific biomarkers of lung tissue injury. Intensive Care Medicine Experimental. 2016;4(Suppl. 1):13. A22. doi: 10.1186/s40635-016-0098-x

Metabonomics identifies early molecular changes associated with progression into postoperative hypoxemia in cardiac surgery patient: a human model that can provide new insights into the pathophysiology of acute lung injury and potentially identify specific biomarkers of lung tissue injury

Conference

Access to Document

AUB Link

Research output

NMR-Based Metabolomics in Kidney and Lung Disease: investigating metabolite biomarkers in a rat chronic kidney disease model and in human lung injury

Cite this