РОЛЬ АКТИВАЦИИ СИМПАТИЧЕСКОЙ НЕРВНОЙ СИСТЕМЫ В РАЗВИТИИ СТРУКТУРНО-ФУНКЦИОНАЛЬНЫХ ИЗМЕНЕНИЙ МИОКАРДА ПРИ СЕРДЕЧНОЙ НЕДОСТАТОЧНОСТИ

Medicine and Dentistry

• Heart Failure 100%
• Isotopes of Calcium 100%
• Cardiac Muscle Cell 100%
• Systolic Dysfunction 66%
• Sarcoplasmic reticulum 66%
• Cyclic AMP Dependent Protein Kinase 33%
• Cytokine 33%
• Cell Growth 33%
• Immediate Early Gene 33%
• Ventricular Hypertrophy 33%
• Cardiac Fibrosis 33%
• Upregulation 33%
• Fibroblast 33%
• Protein Synthesis 33%
• Toxicity 33%
• Programmed Cell Death 33%
• Necrosis 33%
• Matrix Metalloproteinase 33%
• Renin Angiotensin Aldosterone System 33%
• Calcium Transport 33%
• Contractile Function 33%
• Sympathomimetic Drug 33%
• Kinase Signaling 33%
• Ryanodine Receptor 2 33%
• Signal Transduction 33%
• Mitogen-Activated Protein Kinase 33%
• Cell Death 33%
• Growth Factor 33%
• Catecholamine 33%
• Transcription Factors 33%

Keyphrases

• Sympathetic Overactivation 100%
• Overactivation 60%
• Sarcoplasmic Reticulum Calcium 40%
• Calcium Leakage 20%
• Calcium Stores 20%
• Chronic Administration 20%
• Myocardial Development 20%
• SERCA2a 20%
• Early Activation 20%
• Open Probability 20%
• Contractile Proteins 20%
• Adrenergic Agonist 20%
• Cardiac Fibroblasts 20%
• Calcium Handling 20%
• Calcium Uptake 20%
• Hyperphosphorylation 20%
• Collagen Production 20%
• Cross-linked Collagen 20%

Pharmacology, Toxicology and Pharmaceutical Science

• Contractile Protein 33%
• Adrenergic Receptor Stimulating Agent 33%